Activation of telomerase may be a good biomarker for preneoplastic lesions, and associates with cell malignancy. 端粒酶的激活可能是癌前病变的一种生物学标记物，与细胞恶性变的发展有密切关系。
Therefore, to control the activity of telomerase effectively may give a new way for people to surmount c... 因此,有效地控制端粒酶活性,有可能为人类攻克癌症提供一个新的契机。
Results:Oridonin could decrease the activity of telomerase, as well as cause apoptosis and inhibit the growth of K562 cells significantly. 并呈现出明显的量-效与时-效关系。
Activation of telomerase and prolonging of RTF length might be involved in t he process of ginsenoside Rg1 protection against t-BHP-induced senescence in W I-38 cells. 人参皂苷Rg1可能通过激活端粒酶活性和减少端粒长度缩短而发挥其抗t BHP诱导的WI 38细胞衰老作用。
Results The activity of telomerase were 76.2% in gastric carcinoma (GC) group, 48.6% in astrophic gastritis (AG) group, 15.8% in benign gastric ulcers (BGU) group, respectively. 结果胃癌、萎缩性胃炎、良性胃溃疡中端粒酶活性的分别为76.