This relationship is stronger than those observed with fasting glycemia or glycosylated haemoglobin(HbA1c),and persists after adjustment for other vascular risk factors.

• 其机制可能与餐后高血糖时脂质过氧化增强、内皮功能障碍、高凝状态、黏附因子水平升高、炎症、氧化/氮化应激增强等有关。