AIM: In this study, we aimed to explore the alteration and pathophysiological significance of the L-arginine (L-Arg)/NOS/NO pathway in the adventitia of rats with sepsis.

• 目的:观察败血症休克大鼠主动脉外膜L-精氨酸(L-Arg)转运,一氧化氮合酶(NOS)活性和一氧化氮(NO)生成的变化。