A low level of bcl 2 gene expression could be detected in BEC at basic state, and either VIP or EGF stimulated the transcription of bcl 2, which improved the capability of BEC to resist oxidant injury.

• BEC细胞在基础情况下有bcl?2基因的低水平表达，VIP和EGF可促进bcl?2基因转录，增强BEC的抗氧化损伤能力。